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Type 1 diabetes

Type 1 disease modification: preserve, replace, protect

Separate immune modulation, beta-cell replacement, cell protection, and clinically meaningful insulin independence.

2 min readEasy read3 sourcesChecked 30 Sept 2026Preliminary

By The Diabetes Guide editorial project · Updated 30 Sept 2026

On this page
In simple words

Scientists are working on three tasks for Type 1: save the cells that are left, replace the ones that are lost, and protect the new ones.

  1. Step 1: Preserve

    Immune-changing medicines may slow the loss of insulin-making in some people.

    For example: Putting a fence around the last apple trees so they are not chopped down.

  2. Step 2: Replace

    Donor or lab-grown islet cells can make insulin again, for some people, at the moment with medicines that dampen the immune system.

    For example: Planting new trees in the orchard.

  3. Step 3: Protect

    New cells can still be attacked or rejected, so protection is the hardest part.

    For example: Even new trees need a fence, or the same problem returns.

Remember: Solving one task does not solve the other two.

The full story

Want more? Below is the detailed version with the real science words. It is fine to skip it.

Three distinct research tasks

Preserve remaining beta-cell function through immune modulation. Replace lost capacity with donor or manufactured cells. Protect those cells from rejection and autoimmune injury. Achieving one task does not automatically solve the others.

Step by stepA simple model

Type 1 research: solve several problems together

Preserve. Immune modulation can preserve function or delay progression in selected people.

Read every step in a list
  1. Preserve. Immune modulation can preserve function or delay progression in selected people.
  2. Replace. Donor or stem-cell-derived islets can supply insulin-producing cells.
  3. Protect. Immune rejection and recurrent autoimmunity require durable solutions.
  4. Scale safely. Long-term safety, reliable manufacturing, cost and access remain essential.
A research map, not a forecast or promised development timeline. Source: Reichman et al.; New England Journal of Medicine

What human studies establish

The 2026 pediatric Stage 3 teplizumab indication targets decline of insulin production. Zimislecel offers early evidence of functional replacement under immunosuppression. Edited donor islets offer a small, short-term proof of immune protection. Their populations, endpoints and evidence strength differ substantially. 123

What would count as durable progress?

Longer follow-up, independent replication, clinically important benefit, acceptable harms and scalable access matter more than a headline. Read the 2026 evidence review for dates and limitations.

Trace the evidence

Sources and further reading

1.FDA accelerated approval: teplizumab in selected recently diagnosed pediatric Stage 3 T1D (opens in a new tab)

US Food and Drug Administration · 2026 · Regulatory

Strong Evidence
Who was studied, limits and source check

Population: Children aged 8–17 recently diagnosed with Stage 3 T1D

Sample: PROTECT: 328 participants

Limitations: June 12 accelerated approval based on C-peptide preservation, a surrogate. Confirmatory clinical benefit and label conditions matter.

Source checked 2026-09-30. See the original publication for full methods.

2.Stem Cell–Derived, Fully Differentiated Islets for Type 1 Diabetes (opens in a new tab)

Reichman et al.; New England Journal of Medicine · 2025 · Early human study

Preliminary
Who was studied, limits and source check

Population: Selected adults with T1D, severe hypoglycemia and impaired awareness

Sample: 14 with ≥12-month follow-up; 12 at full dose

Limitations: Small, uncontrolled interim analysis; required immunosuppression; serious adverse events and deaths occurred. Not a routine cure.

Source checked 2026-09-30. See the original publication for full methods.

3.Survival of Transplanted Allogeneic Beta Cells with No Immunosuppression (opens in a new tab)

Carlsson et al.; New England Journal of Medicine · 2025 · Early human study

Experimental
Who was studied, limits and source check

Population: One adult with long-standing T1D

Sample: 1 participant; 12-week reported follow-up

Limitations: Single participant, low cell dose and short follow-up. Does not demonstrate generalizable insulin independence.

Source checked 2026-09-30. See the original publication for full methods.

Source checking is an editorial literature check, not independent medical review. This page is for learning. It cannot diagnose you or make a treatment plan. Evidence labels describe the cited claims, not the whole topic.

What does “Preliminary” mean?

Early results. They need to be repeated. Like a first test drive of a new car.

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